References
[1]. K. M. Flegal, M. D. Carroll, B. K. Kit, et al., "Prevalence of obesity and trends in the distribution of body mass index among US adults, 1999-2010," Jama, 307(5), 491-497 (2012).
[2]. M. Ng, T. Fleming, M. Robinson, et al., "Global, regional, and national prevalence of overweight and obesity in children and adults during 1980–2013: a systematic analysis for the Global Burden of Disease Study 2013," The lancet, 384(9945), 766-781 (2014).
[3]. E. Han,L. M. Powell, "Consumption patterns of sugar-sweetened beverages in the United States," Journal of the Academy of Nutrition and Dietetics, 113(1), 43-53 (2013).
[4]. M. S. Mesirow,J. A. Welsh, "Changing beverage consumption patterns have resulted in fewer liquid calories in the diets of US children: National Health and Nutrition Examination Survey 2001-2010," Journal of the Academy of Nutrition and Dietetics, 115(4), 559-566. e4 (2015).
[5]. J. C. de Ruyter, M. R. Olthof, J. C. Seidell, et al., "A trial of sugar-free or sugar-sweetened beverages and body weight in children," New England Journal of Medicine, 367(15), 1397-1406 (2012).
[6]. D. F. Tate, G. Turner-McGrievy, E. Lyons, et al., "Replacing caloric beverages with water or diet beverages for weight loss in adults: main results of the C hoose H ealthy O pt i ons C onsciously E veryday (CHOICE) randomized clinical trial," The American journal of clinical nutrition, 95(3), 555-563 (2012).
[7]. K. Mathur, R. K. Agrawal, S. Nagpure, et al., "Effect of artificial sweeteners on insulin resistance among type-2 diabetes mellitus patients," Journal of family medicine and primary care, 9(1), 69-71 (2020).
[8]. J. Suez, T. Korem, D. Zeevi, et al., "Artificial sweeteners induce glucose intolerance by altering the gut microbiota," Nature, 514(7521), 181-186 (2014).
[9]. E. E. Canfora, R. C. Meex, K. Venema, et al., "Gut microbial metabolites in obesity, NAFLD and T2DM," Nature Reviews Endocrinology, 15(5), 261-273 (2019).
[10]. S. M. Jandhyala, R. Talukdar, C. Subramanyam, et al., "Role of the normal gut microbiota," World journal of gastroenterology: WJG, 21(29), 8787 (2015).
[11]. P. G. Farup, S. Lydersen,J. Valeur, "Are nonnutritive sweeteners obesogenic? Associations between diet, faecal microbiota, and short-chain fatty acids in morbidly obese subjects," Journal of Obesity, 2019 (2019).
[12]. C. L. Frankenfeld, M. Sikaroodi, E. Lamb, et al., "High-intensity sweetener consumption and gut microbiome content and predicted gene function in a cross-sectional study of adults in the United States," Annals of epidemiology, 25(10), 736-742. e4 (2015).
[13]. B. A. Magnuson, M. C. Carakostas, N. H. Moore, et al., "Biological fate of low-calorie sweeteners," Nutrition Reviews, 74(11), 670-689 (2016).
[14]. S. Chattopadhyay, U. Raychaudhuri,R. Chakraborty, "Artificial sweeteners–a review," Journal of food science and technology, 51(4), 611-621 (2014).
[15]. Omran, G. Ahearn, D. Bowers, et al., "Metabolic effects of sucralose on environmental bacteria," Journal of toxicology, 2013 (2013).
[16]. D. Sonagra, S. M. Biradar, K. Dattatreya, et al., "Normal pregnancy-a state of insulin resistance," Journal of clinical and diagnostic research: JCDR, 8(11), CC01 (2014).
[17]. P. G. Ovesen, J. Fuglsang, M. B. Andersen, et al., "Temporal trends in gestational diabetes prevalence, treatment, and outcomes at Aarhus University Hospital, Skejby, between 2004 and 2016," Journal of diabetes research, 2018 (2018).
[18]. L. L. Kurtzhals, S. K. Nørgaard, A. L. Secher, et al., "The impact of restricted gestational weight gain by dietary intervention on fetal growth in women with gestational diabetes mellitus," Diabetologia, 61(12), 2528-2538 (2018).
[19]. F. Nicolì, A. Prete, F. Citro, et al., "Use of non-nutritive-sweetened soft drink and risk of gestational diabetes," Diabetes Research and Clinical Practice, 178, 108943 (2021).
[20]. E. Szoke, M. Z. Shrayyef, S. Messing, et al., "Effect of aging on glucose homeostasis: accelerated deterioration of β-cell function in individuals with impaired glucose tolerance," Diabetes care, 31(3), 539-543 (2008).
Cite this article
Han,Y. (2023). Analysis of The Effects of Artificial Sweeteners on Diabetes and Obesity. Theoretical and Natural Science,4,32-38.
Data availability
The datasets used and/or analyzed during the current study will be available from the authors upon reasonable request.
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References
[1]. K. M. Flegal, M. D. Carroll, B. K. Kit, et al., "Prevalence of obesity and trends in the distribution of body mass index among US adults, 1999-2010," Jama, 307(5), 491-497 (2012).
[2]. M. Ng, T. Fleming, M. Robinson, et al., "Global, regional, and national prevalence of overweight and obesity in children and adults during 1980–2013: a systematic analysis for the Global Burden of Disease Study 2013," The lancet, 384(9945), 766-781 (2014).
[3]. E. Han,L. M. Powell, "Consumption patterns of sugar-sweetened beverages in the United States," Journal of the Academy of Nutrition and Dietetics, 113(1), 43-53 (2013).
[4]. M. S. Mesirow,J. A. Welsh, "Changing beverage consumption patterns have resulted in fewer liquid calories in the diets of US children: National Health and Nutrition Examination Survey 2001-2010," Journal of the Academy of Nutrition and Dietetics, 115(4), 559-566. e4 (2015).
[5]. J. C. de Ruyter, M. R. Olthof, J. C. Seidell, et al., "A trial of sugar-free or sugar-sweetened beverages and body weight in children," New England Journal of Medicine, 367(15), 1397-1406 (2012).
[6]. D. F. Tate, G. Turner-McGrievy, E. Lyons, et al., "Replacing caloric beverages with water or diet beverages for weight loss in adults: main results of the C hoose H ealthy O pt i ons C onsciously E veryday (CHOICE) randomized clinical trial," The American journal of clinical nutrition, 95(3), 555-563 (2012).
[7]. K. Mathur, R. K. Agrawal, S. Nagpure, et al., "Effect of artificial sweeteners on insulin resistance among type-2 diabetes mellitus patients," Journal of family medicine and primary care, 9(1), 69-71 (2020).
[8]. J. Suez, T. Korem, D. Zeevi, et al., "Artificial sweeteners induce glucose intolerance by altering the gut microbiota," Nature, 514(7521), 181-186 (2014).
[9]. E. E. Canfora, R. C. Meex, K. Venema, et al., "Gut microbial metabolites in obesity, NAFLD and T2DM," Nature Reviews Endocrinology, 15(5), 261-273 (2019).
[10]. S. M. Jandhyala, R. Talukdar, C. Subramanyam, et al., "Role of the normal gut microbiota," World journal of gastroenterology: WJG, 21(29), 8787 (2015).
[11]. P. G. Farup, S. Lydersen,J. Valeur, "Are nonnutritive sweeteners obesogenic? Associations between diet, faecal microbiota, and short-chain fatty acids in morbidly obese subjects," Journal of Obesity, 2019 (2019).
[12]. C. L. Frankenfeld, M. Sikaroodi, E. Lamb, et al., "High-intensity sweetener consumption and gut microbiome content and predicted gene function in a cross-sectional study of adults in the United States," Annals of epidemiology, 25(10), 736-742. e4 (2015).
[13]. B. A. Magnuson, M. C. Carakostas, N. H. Moore, et al., "Biological fate of low-calorie sweeteners," Nutrition Reviews, 74(11), 670-689 (2016).
[14]. S. Chattopadhyay, U. Raychaudhuri,R. Chakraborty, "Artificial sweeteners–a review," Journal of food science and technology, 51(4), 611-621 (2014).
[15]. Omran, G. Ahearn, D. Bowers, et al., "Metabolic effects of sucralose on environmental bacteria," Journal of toxicology, 2013 (2013).
[16]. D. Sonagra, S. M. Biradar, K. Dattatreya, et al., "Normal pregnancy-a state of insulin resistance," Journal of clinical and diagnostic research: JCDR, 8(11), CC01 (2014).
[17]. P. G. Ovesen, J. Fuglsang, M. B. Andersen, et al., "Temporal trends in gestational diabetes prevalence, treatment, and outcomes at Aarhus University Hospital, Skejby, between 2004 and 2016," Journal of diabetes research, 2018 (2018).
[18]. L. L. Kurtzhals, S. K. Nørgaard, A. L. Secher, et al., "The impact of restricted gestational weight gain by dietary intervention on fetal growth in women with gestational diabetes mellitus," Diabetologia, 61(12), 2528-2538 (2018).
[19]. F. Nicolì, A. Prete, F. Citro, et al., "Use of non-nutritive-sweetened soft drink and risk of gestational diabetes," Diabetes Research and Clinical Practice, 178, 108943 (2021).
[20]. E. Szoke, M. Z. Shrayyef, S. Messing, et al., "Effect of aging on glucose homeostasis: accelerated deterioration of β-cell function in individuals with impaired glucose tolerance," Diabetes care, 31(3), 539-543 (2008).